AP Psychology Mental and Physical Health — Worked Answer Explanations
Unit 5 · 12 questions explained
Below is a complete answer key for our AP Psychology Mental and Physical Health practice questions. For each question you'll find the correct choice, a full written explanation of how to get there, and — for every wrong answer — a short note on exactly why it's tempting and where it goes wrong. Reading these straight through is one of the fastest ways to find the gaps in a unit before exam day.
Prefer to test yourself first? Take the timed Mental and Physical Health practice test and come back here to review, or head back to the Mental and Physical Health unit overview.
- Question 1 · Easy
The DSM-5 is the primary tool used by mental health professionals in the United States to:
- APrescribe medication dosages for psychiatric disordersWhy not A: Medication prescribing is guided by pharmacology and clinical judgment, not the DSM-5, which is a diagnostic classification system.
- BClassify and diagnose psychological disorders using standardized criteriaCorrect
- CAssess intelligence and academic achievement levelsWhy not C: Intelligence and achievement testing use separate instruments (e.g., WISC, WAIS, Stanford-Binet); the DSM-5 is for diagnosing mental disorders.
- DDetermine the causes of psychological disorders through biological testingWhy not D: The DSM-5 uses descriptive criteria (symptoms, duration, functional impairment) rather than biological tests to classify disorders.
ExplanationThe Diagnostic and Statistical Manual of Mental Disorders, Fifth Edition (DSM-5), published by the American Psychiatric Association, provides standardized diagnostic criteria for mental disorders. Clinicians use it to ensure consistent diagnosis across practitioners and settings, facilitate communication, guide treatment planning, and qualify patients for insurance coverage. The DSM-5 shifted from a multiaxial system to a single-axis approach and incorporated dimensional assessments.
Key takeawayDSM-5: standardized classification and diagnostic criteria for psychological disorders in clinical and research settings.
- A
- Question 2 · Easy
Which of the following best describes the hallmark features of major depressive disorder (MDD) according to DSM-5 criteria?
- AAlternating episodes of intense elation (mania) and deep depression lasting at least two yearsWhy not A: Alternating mania and depression describes bipolar disorder; two-year duration is the criterion for cyclothymia or persistent depressive disorder.
- BPersistent depressed mood or loss of interest for at least two weeks, causing significant impairmentCorrect
- CRecurrent unexpected panic attacks with persistent worry about future attacksWhy not C: Recurrent unexpected panic attacks with anticipatory anxiety describe panic disorder, not major depressive disorder.
- DIntrusive thoughts and compulsive rituals that reduce anxietyWhy not D: Intrusive obsessions and compulsive rituals are the hallmarks of obsessive-compulsive disorder (OCD), not depression.
ExplanationDSM-5 criteria for Major Depressive Disorder require five or more depressive symptoms (e.g., depressed mood, loss of interest/anhedonia, sleep changes, fatigue, concentration difficulties, feelings of worthlessness, suicidal ideation) present during the same two-week period, with at least one of the symptoms being depressed mood or anhedonia. Symptoms must cause clinically significant distress or functional impairment and not be attributable to substances or another medical condition.
Key takeawayMDD hallmarks: depressed mood or anhedonia for 2+ weeks with 5+ depressive symptoms causing significant impairment.
- A
- Question 3 · Easy
A therapist treats a patient with anxiety disorder by teaching deep breathing and relaxation, then gradually exposing the patient to feared stimuli from least to most anxiety-provoking. This technique is called:
- AFlooding (implosive therapy)Why not A: Flooding exposes the patient to the most feared stimulus immediately and intensely without a gradual hierarchy; the description specifically mentions gradual exposure.
- BAversion therapyWhy not B: Aversion therapy pairs an undesired behavior with an unpleasant stimulus to reduce it (e.g., treating alcohol addiction with nausea-inducing drugs); it does not involve building a fear hierarchy.
- CSystematic desensitizationCorrect
- DToken economyWhy not D: Token economy is an operant conditioning technique using symbolic rewards for desired behaviors; it is not an exposure-based anxiety treatment.
ExplanationSystematic desensitization, developed by Joseph Wolpe, is a behavioral therapy based on classical conditioning principles. It combines deep relaxation training with gradual exposure to feared stimuli through an anxiety hierarchy. The relaxation response becomes conditioned to the feared stimulus, replacing the anxiety response (counterconditioning). It is among the most effective evidence-based treatments for phobias, PTSD, and anxiety disorders.
Key takeawaySystematic desensitization: counterconditioning anxiety through pairing relaxation with a gradual hierarchy of feared stimuli.
- A
- Question 4 · Easy
Hans Selye's General Adaptation Syndrome (GAS) describes the body's response to prolonged stress in three sequential stages. In the correct order, these stages are:
- AResistance → Alarm → ExhaustionWhy not A: This places resistance before alarm, reversing the first two stages; the initial response to a stressor is always the alarm reaction.
- BAlarm → Exhaustion → ResistanceWhy not B: This incorrectly places exhaustion before resistance; the body first mounts coping resources (resistance) before resources are depleted (exhaustion).
- CAlarm → Resistance → ExhaustionCorrect
- DExhaustion → Alarm → ResistanceWhy not D: Exhaustion is the final stage when coping resources are depleted; it does not precede the initial alarm response.
ExplanationSelye's GAS describes a three-stage biological stress response: (1) Alarm — the initial fight-or-flight activation (HPA axis, sympathetic nervous system, cortisol surge); (2) Resistance — the body mobilizes and attempts to cope with the ongoing stressor while returning to a more normal state; (3) Exhaustion — prolonged stress depletes physiological resources, increasing vulnerability to illness, depression, and organ damage. The GAS explains why chronic stress harms physical health.
Key takeawaySelye's GAS sequence: Alarm → Resistance → Exhaustion; exhaustion from chronic stress leads to health breakdown.
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- Question 5 · Medium
A client with post-traumatic stress disorder works with a therapist who uses EMDR (Eye Movement Desensitization and Reprocessing). The therapist also helps the client identify distorted thoughts like 'I am permanently ruined.' This cognitive component is most closely associated with which therapy approach?
- AClient-centered (humanistic) therapyWhy not A: Client-centered therapy (Rogers) emphasizes empathy, unconditional positive regard, and genuine understanding — not cognitive restructuring of distorted thoughts.
- BPsychoanalytic therapyWhy not B: Psychoanalytic therapy focuses on unconscious conflicts, free association, and dream analysis — not the direct identification and challenging of cognitive distortions.
- CCognitive-behavioral therapy (CBT)Correct
- DDrug therapy (pharmacotherapy)Why not D: Drug therapy involves biological interventions (medications) to reduce symptoms; it does not involve talking about thought patterns.
ExplanationCognitive-behavioral therapy (CBT), integrating Beck's cognitive therapy and behavioral techniques, targets both maladaptive cognitions (distorted beliefs like 'I am permanently ruined') and behaviors. For PTSD, CBT-based approaches like Cognitive Processing Therapy specifically help patients identify and restructure trauma-related cognitive distortions. The behavioral component might include exposure exercises. CBT is the most empirically supported psychological treatment across multiple disorders.
Key takeawayCBT targets cognitive distortions (thoughts) and maladaptive behaviors simultaneously; it is the most evidence-based psychotherapy for many disorders.
- A
- Question 6 · Medium
A patient is diagnosed with schizophrenia. Which cluster of symptoms is categorized as 'positive symptoms' in DSM-5?
- AFlat affect, alogia (poverty of speech), and avolition (lack of motivation)Why not A: These are negative symptoms — the absence or reduction of normal functions (emotion, speech, motivation). Negative symptoms are harder to treat and have a worse prognosis.
- BHallucinations, delusions, and disorganized speechCorrect
- CCognitive deficits in attention, working memory, and processing speedWhy not C: Cognitive impairments are recognized features of schizophrenia but are categorized separately from the positive/negative symptom framework.
- DDepressed mood, sleep disturbances, and suicidal ideationWhy not D: These are depressive symptoms; when they occur alongside schizophrenia features, the diagnosis may be schizoaffective disorder, but they are not positive symptoms of schizophrenia per se.
ExplanationIn schizophrenia, positive symptoms are excesses or additions to normal functioning — things that are present but should not be: hallucinations (false perceptions, most often auditory), delusions (fixed false beliefs, e.g., persecution, grandiosity), and disorganized speech/behavior. Negative symptoms are deficits — things that are absent but should be present: flat affect, alogia, avolition, anhedonia, asociality. Positive symptoms respond better to antipsychotic medications; negative symptoms are more persistent.
Key takeawaySchizophrenia positive symptoms: hallucinations, delusions, disorganized speech (excesses); negative symptoms: flat affect, avolition, alogia (deficits).
- A
- Question 7 · Medium
A psychiatrist prescribes an SSRI (selective serotonin reuptake inhibitor) to a patient with depression. At the synaptic level, this medication works by:
- ABlocking dopamine receptors on the postsynaptic neuronWhy not A: Blocking dopamine receptors is the mechanism of antipsychotics (e.g., haloperidol) used to treat schizophrenia, not SSRIs for depression.
- BIncreasing serotonin synthesis in the presynaptic neuronWhy not B: SSRIs do not increase synthesis; they prevent reuptake. The drug acts on transport proteins, not on biosynthesis enzymes.
- CBlocking the reuptake transporter so serotonin remains in the synapse longerCorrect
- DMimicking serotonin by binding directly to postsynaptic serotonin receptorsWhy not D: Agonist drugs mimic neurotransmitters by binding receptors, but SSRIs work on the presynaptic reuptake mechanism, not by acting as serotonin mimics.
ExplanationSSRIs (e.g., fluoxetine/Prozac, sertraline/Zoloft) inhibit the serotonin reuptake transporter (SERT) on the presynaptic neuron. Normally, after serotonin is released into the synapse and acts on receptors, it is taken back up into the sending neuron for recycling. By blocking this transporter, SSRIs allow serotonin to remain in the synaptic cleft longer and stimulate postsynaptic receptors more persistently, effectively increasing serotonergic transmission.
Key takeawaySSRIs block serotonin reuptake transporters, increasing serotonin availability in the synapse — the mechanism underlying antidepressant effects.
- A
- Question 8 · Medium
Albert Ellis developed Rational Emotive Behavior Therapy (REBT) based on his ABC model. In this model, 'B' stands for:
- ABehavior — the observable actions taken in response to eventsWhy not A: Behavior is important in CBT broadly, but in Ellis's ABC model, B specifically refers to beliefs, not behaviors.
- BBiology — the physiological arousal accompanying emotional distressWhy not B: Ellis's model is cognitive-focused; biology is not one of the three components of the ABC framework.
- CBeliefs — the rational or irrational thoughts about the activating eventCorrect
- DBackground — the early childhood experiences shaping personalityWhy not D: Background history is a psychodynamic focus; Ellis explicitly argued against dwelling on past causes — REBT targets current irrational beliefs.
ExplanationIn Ellis's ABC model: A = Activating event (the situation); B = Beliefs (irrational or rational thoughts about the event); C = Consequences (emotional and behavioral outcomes). Crucially, Ellis argued that A does not directly cause C — B causes C. People disturb themselves with irrational beliefs ('I must be loved by everyone or I am worthless'). REBT teaches clients to dispute (D) these irrational beliefs to achieve a new emotional effect (E). This is the cognitive foundation for modern CBT.
Key takeawayEllis's REBT ABC model: A (activating event) → B (beliefs) → C (consequences). Irrational beliefs (B) cause emotional distress, not the events themselves.
- A
- Question 9 · Hard
Research on optimism and health outcomes consistently shows that dispositional optimists have better immune function and recover more quickly from surgery. The biopsychosocial model best explains this finding by noting that:
- AOptimism directly increases the number of T-cells produced, independent of behavior or psychology.Why not A: The relationship is mediated by behavior and psychological factors; optimism does not directly and independently control immune cell production through a simple causal pathway.
- BPsychological states influence biological health outcomes through behavioral and neuroendocrine pathways.Correct
- CHealth outcomes are determined entirely by genetics and biological factors regardless of psychological state.Why not C: This describes a purely biomedical model, which the biopsychosocial model explicitly rejects by incorporating psychological and social factors.
- DOptimism is a learned behavior reinforced by health professionals during clinical interactions.Why not D: This operant conditioning explanation does not account for the psychoneuroimmunology research showing how cognitive-emotional states affect biology.
ExplanationThe biopsychosocial model (Engel) holds that health and illness are determined by biological, psychological, and social factors interacting together. Optimism improves health through multiple pathways: cognitively (reducing appraisal of threat), behaviorally (better health behaviors, treatment adherence), and neuroendocrine-immune (lower cortisol, healthier HPA axis response). Psychoneuroimmunology — the study of how psychological states affect immune function — directly bridges the psychological and biological levels.
Key takeawayBiopsychosocial model: psychological states (optimism/pessimism) affect physical health through behavioral, neural, and hormonal pathways.
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- Question 10 · Hard
A clinical psychologist is asked whether a particular psychological test measures what it claims to measure — for example, whether a test labeled 'social anxiety scale' actually captures social anxiety and not general neuroticism. The psychologist is evaluating the test's:
- AReliabilityWhy not A: Reliability refers to the consistency of the test's results across time and administrations — not whether it measures the right construct.
- BStandardizationWhy not B: Standardization means the test is administered and scored uniformly across participants; it is a procedural quality, not a measure of whether the test captures the intended construct.
- CConstruct validityCorrect
- DPredictive validityWhy not D: Predictive validity asks whether the test predicts a future criterion (e.g., does the test predict therapy dropout rates?) — not whether it correctly measures the underlying theoretical construct.
ExplanationConstruct validity is the degree to which a test actually measures the theoretical construct it purports to measure. In this case: does the social anxiety scale capture social anxiety specifically, rather than a broader trait like neuroticism? Establishing construct validity involves convergent validity (correlates with related measures) and discriminant validity (does not correlate with unrelated constructs). Reliability is necessary but not sufficient for validity — a test can be consistently wrong.
Key takeawayConstruct validity: does the test measure the specific theoretical construct it claims to measure? Reliable ≠ valid.
- A
- Question 11 · Hard
In the diathesis-stress model of psychological disorders, a person develops a disorder when:
- AThey are exposed to a severe stressor with no prior biological vulnerabilityWhy not A: A stressor alone, without a pre-existing vulnerability (diathesis), is insufficient in this model; both components are required.
- BA genetic or biological vulnerability (diathesis) is activated by sufficient environmental stressCorrect
- CTheir neurotransmitter imbalances reach a clinical threshold regardless of environmental factorsWhy not C: This describes a purely biological model; the diathesis-stress model is explicitly interactive, requiring an environmental trigger to activate the vulnerability.
- DThey learn maladaptive behaviors through repeated classical conditioningWhy not D: Classical conditioning is a behavioral explanation for specific learned fear responses, not the diathesis-stress framework that explains disorder onset via vulnerability × environment interaction.
ExplanationThe diathesis-stress model proposes that psychological disorders develop through an interaction between a predisposing vulnerability (diathesis — genetic, neurobiological, or psychological) and environmental stressors. Neither the diathesis nor the stress alone is sufficient — the disorder emerges when stress exceeds the threshold set by the diathesis. This model explains why not everyone exposed to trauma develops PTSD, and why some people with genetic risk for depression never develop it without significant life stress.
Key takeawayDiathesis-stress model: disorder = predisposing vulnerability (diathesis) × environmental stressor; neither alone is sufficient.
- A
- Question 12 · Hard
A study finds that cancer patients who participate in a structured social support group have significantly longer survival times than those who do not. Critics argue the study may confound social support with the effects of patients discussing healthier behaviors. To control for this alternative explanation, the researchers should:
- AUse a larger sample size to increase statistical power.Why not A: A larger sample increases the ability to detect real effects but does not eliminate the confound — the alternative explanation (health behavior discussion) remains uncontrolled regardless of sample size.
- BAdd a control group that meets socially but discusses topics unrelated to health behaviors.Correct
- CMeasure survival time using a blinded assessor to prevent experimenter bias.Why not C: Blinding assessors controls for measurement bias on the dependent variable but does not separate social support from health-behavior discussion as the active ingredient.
- DSurvey participants about whether they found the groups helpful.Why not D: Self-report of perceived helpfulness does not isolate the mechanism of action; both groups could find their respective groups helpful for different reasons.
ExplanationTo isolate the effect of social support per se from the confound of health behavior discussions, the researchers need an active control group that receives the same social contact (controlling for social support benefits) without the health behavior content. This is a dismantling strategy: the experimental group gets full treatment; the control group gets social contact minus the specific ingredient under investigation. If the full-treatment group still outperforms the control, health discussion must be adding something beyond mere social contact.
Key takeawayTo isolate a treatment component, use an active control that matches all other features except the ingredient being tested — a dismantling design.
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